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A Novel Isotope-labeled Small Molecule Probe CC12 for Anti-glioma via  Suppressing LYN-mediated Progression and Activating Apoptosis Pathways
A Novel Isotope-labeled Small Molecule Probe CC12 for Anti-glioma via Suppressing LYN-mediated Progression and Activating Apoptosis Pathways

Blood-brain barrier disruption defines the extracellular metabolome of live  human high-grade gliomas | Communications Biology
Blood-brain barrier disruption defines the extracellular metabolome of live human high-grade gliomas | Communications Biology

CCL2-Expressing Astrocytes Mediate the Extravasation of T Lymphocytes in  the Brain. Evidence from Patients with Glioma and Experimental Models In  Vivo | PLOS ONE
CCL2-Expressing Astrocytes Mediate the Extravasation of T Lymphocytes in the Brain. Evidence from Patients with Glioma and Experimental Models In Vivo | PLOS ONE

Frontiers | Glioma-derived CCL2 and CCL7 mediate migration of immune  suppressive CCR2+/CX3CR1+ M-MDSCs into the tumor microenvironment in a  redundant manner
Frontiers | Glioma-derived CCL2 and CCL7 mediate migration of immune suppressive CCR2+/CX3CR1+ M-MDSCs into the tumor microenvironment in a redundant manner

Immune cell trafficking across the blood-brain barrier in the absence and  presence of neuroinflammation in: Vascular Biology Volume 2 Issue 1 (2020)
Immune cell trafficking across the blood-brain barrier in the absence and presence of neuroinflammation in: Vascular Biology Volume 2 Issue 1 (2020)

Neuroinflammatory mechanisms of blood-brain barrier damage in ischemic  stroke | American Journal of Physiology-Cell Physiology
Neuroinflammatory mechanisms of blood-brain barrier damage in ischemic stroke | American Journal of Physiology-Cell Physiology

CCL2-Expressing Astrocytes Mediate the Extravasation of T Lymphocytes in  the Brain. Evidence from Patients with Glioma and Experimental Models In  Vivo | PLOS ONE
CCL2-Expressing Astrocytes Mediate the Extravasation of T Lymphocytes in the Brain. Evidence from Patients with Glioma and Experimental Models In Vivo | PLOS ONE

Frontiers | Deciphering immune microenvironment and cell evasion mechanisms  in human gliomas
Frontiers | Deciphering immune microenvironment and cell evasion mechanisms in human gliomas

Glioma-derived exosomes hijack the blood–brain barrier to facilitate  nanocapsule delivery via LCN2 - ScienceDirect
Glioma-derived exosomes hijack the blood–brain barrier to facilitate nanocapsule delivery via LCN2 - ScienceDirect

Crosstalk among BC cells, glial cells, and the brain microenvironment.... |  Download Scientific Diagram
Crosstalk among BC cells, glial cells, and the brain microenvironment.... | Download Scientific Diagram

Pharmaceutics | Free Full-Text | Neuroimmunomodulatory Properties of  Flavonoids and Derivates: A Potential Action as Adjuvants for the Treatment  of Glioblastoma
Pharmaceutics | Free Full-Text | Neuroimmunomodulatory Properties of Flavonoids and Derivates: A Potential Action as Adjuvants for the Treatment of Glioblastoma

IJMS | Free Full-Text | Microglia and the Blood–Brain Barrier: An  External Player in Acute and Chronic Neuroinflammatory Conditions
IJMS | Free Full-Text | Microglia and the Blood–Brain Barrier: An External Player in Acute and Chronic Neuroinflammatory Conditions

Remodeling the blood–brain barrier microenvironment by natural products for  brain tumor therapy - ScienceDirect
Remodeling the blood–brain barrier microenvironment by natural products for brain tumor therapy - ScienceDirect

IJMS | Free Full-Text | Microglia and the Blood–Brain Barrier: An  External Player in Acute and Chronic Neuroinflammatory Conditions
IJMS | Free Full-Text | Microglia and the Blood–Brain Barrier: An External Player in Acute and Chronic Neuroinflammatory Conditions

The CCL2-CCR2 astrocyte-cancer cell axis in tumor extravasation at the  brain | Science Advances
The CCL2-CCR2 astrocyte-cancer cell axis in tumor extravasation at the brain | Science Advances

Frontiers | Origin, activation, and targeted therapy of glioma-associated  macrophages
Frontiers | Origin, activation, and targeted therapy of glioma-associated macrophages

CCL2 (C-C Motif Chemokine Ligand 2) Biomarker Responses in Central Versus  Peripheral Compartments After Focal Cerebral Ischemia | Stroke
CCL2 (C-C Motif Chemokine Ligand 2) Biomarker Responses in Central Versus Peripheral Compartments After Focal Cerebral Ischemia | Stroke

Increased Antiangiogenic Effect by Blocking CCL2-dependent Macrophages in a  Rodent Glioblastoma Model: Correlation Study with Dynamic Susceptibility  Contrast Perfusion MRI | Scientific Reports
Increased Antiangiogenic Effect by Blocking CCL2-dependent Macrophages in a Rodent Glioblastoma Model: Correlation Study with Dynamic Susceptibility Contrast Perfusion MRI | Scientific Reports

Frontiers | Study of BBB Dysregulation in Neuropathogenicity Using  Integrative Human Model of Blood–Brain Barrier
Frontiers | Study of BBB Dysregulation in Neuropathogenicity Using Integrative Human Model of Blood–Brain Barrier

Increased Antiangiogenic Effect by Blocking CCL2-dependent Macrophages in a  Rodent Glioblastoma Model: Correlation Study with Dynamic Susceptibility  Contrast Perfusion MRI | Scientific Reports
Increased Antiangiogenic Effect by Blocking CCL2-dependent Macrophages in a Rodent Glioblastoma Model: Correlation Study with Dynamic Susceptibility Contrast Perfusion MRI | Scientific Reports

Blood-brain barrier disruption defines the extracellular metabolome of live  human high-grade gliomas | Communications Biology
Blood-brain barrier disruption defines the extracellular metabolome of live human high-grade gliomas | Communications Biology

Frontiers | Oncolytic Viro-Immunotherapy: An Emerging Option in the  Treatment of Gliomas
Frontiers | Oncolytic Viro-Immunotherapy: An Emerging Option in the Treatment of Gliomas

Smart Nanomedicine to Enable Crossing Blood–Brain Barrier Delivery of  Checkpoint Blockade Antibody for Immunotherapy of Glioma | ACS Nano
Smart Nanomedicine to Enable Crossing Blood–Brain Barrier Delivery of Checkpoint Blockade Antibody for Immunotherapy of Glioma | ACS Nano

Frontiers | CCL2 Overexpression in the Brain Promotes Glial Activation and  Accelerates Tau Pathology in a Mouse Model of Tauopathy
Frontiers | CCL2 Overexpression in the Brain Promotes Glial Activation and Accelerates Tau Pathology in a Mouse Model of Tauopathy

The blood–tumour barrier in cancer biology and therapy | Nature Reviews  Clinical Oncology
The blood–tumour barrier in cancer biology and therapy | Nature Reviews Clinical Oncology

Pericytes augment glioblastoma cell resistance to temozolomide through  CCL5-CCR5 paracrine signaling | Cell Research
Pericytes augment glioblastoma cell resistance to temozolomide through CCL5-CCR5 paracrine signaling | Cell Research